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Mitochondria, Oxidative Stress and Innate Immunity 期刊论文 WOS高被引论文

编号：

4e05a68b-3ed3-486d-a450-f25b325c7aa8
作者：

Chen, Yuxin(陈雨欣)#*^[1]Zhou, Zhongyang^[2];Min, Wang*^[2,3,4]
语种：

英文
期刊：

FRONTIERS IN PHYSIOLOGY ISSN：1664-042X 2018 年 9 卷 ; OCT 18
收录：
关键词：

mitochondria innate and adaptive immune response inflammasome ROS thioredoxin
摘要：

Canonical functions of mitochondria include the regulation of cellular survival, orchestration of anabolic and metabolic pathways, as well as reactive oxygen species (ROS) signaling. Recent discoveries, nevertheless, have demonstrated that mitochondria are also critical elements to stimulate innate immune signaling cascade that is able to intensify the inflammation upon cytotoxic stimuli beyond microbial infection. Here we review the expanding research field of mitochondria and oxidative stress in innate immune system to highlight the new mechanistic insights and discuss the pathological relevance of mitochondrial dysregulation induced aberrant innate immune responses in a growing list of sterile inflammatory diseases.
推荐引用方式
GB/T 7714：

Chen Yuxin,Zhou Zhongyang,Min Wang, et al. Mitochondria, Oxidative Stress and Innate Immunity [J].FRONTIERS IN PHYSIOLOGY,2018,9.
APA：

Chen Yuxin,Zhou Zhongyang,Min Wang.(2018).Mitochondria, Oxidative Stress and Innate Immunity .FRONTIERS IN PHYSIOLOGY,9.
MLA：

Chen Yuxin, et al. "Mitochondria, Oxidative Stress and Innate Immunity" .FRONTIERS IN PHYSIOLOGY 9(2018).
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